Studying the mechanisms of DNA damage and repair in lung cancer
Table Of Contents
<p>1. Introduction<br> 1.1 Background<br> 1.2 Objectives<br>2. DNA Damage and Genomic Instability in Lung Cancer<br> 2.1 Types of DNA Damage in Lung Cancer<br> 2.2 Causes of DNA Damage in Lung Cancer<br> 2.3 Consequences of Genomic Instability<br>3. DNA Repair Pathways in Lung Cancer<br> 3.1 Base Excision Repair (BER)<br> 3.2 Nucleotide Excision Repair (NER)<br> 3.3 Mismatch Repair (MMR)<br> 3.4 Homologous Recombination (HR)<br> 3.5 Non-Homologous End Joining (NHEJ)<br>4. DNA Repair Deficiencies in Lung Cancer<br> 4.1 Mutations in DNA Repair Genes<br> 4.2 Epigenetic Regulation of DNA Repair<br> 4.3 Microenvironmental Factors and DNA Repair<br>5. Impact of DNA Damage and Repair on Lung Cancer Progression<br> 5.1 DNA Repair and Chemoresistance<br> 5.2 DNA Repair and Radioresistance<br> 5.3 DNA Repair and Immunotherapy Response<br></p>
Project Abstract
Lung cancer is a complex disease characterized by genetic alterations and DNA damage. This project aims to investigate the mechanisms of DNA damage and repair in lung cancer, focusing on the role of various DNA repair pathways, the impact of DNA damage on genomic stability, and the potential for targeted therapies. By understanding the underlying biochemistry of DNA repair in lung cancer, we can develop novel strategies for diagnosis, prognosis, and treatment.
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